Harrison, M.M., Jenkins, B.V., O'Connor-Giles, K.M., and Wildonger, J. (2014). A CRISPR view of development. Genes & development 28, 1859-1872.
Gratz, S.J*., Ukken, F.P*., Rubinstein, C.D., Thiede, G., Donohue, L.K., Cummings, A.M., and O'Connor-Giles, K.M. (2014). Highly Specific and Efficient CRISPR/Cas9-Catalyzed Homology-Directed Repair in Drosophila. Genetics 196, 961-971.
*These authors contributed equally.
Gratz, S.J., Wildonger, J., Harrison, M.M., and O'Connor-Giles, K.M. (2013b). CRISPR/Cas9-mediated genome engineering and the promise of designer flies on demand. Fly (Austin) 7.
Gratz, S.J., Cummings, A.M., Nguyen, J.N., Hamm, D.C., Donohue, L.K., Harrison, M.M., Wildonger, J., and O'Connor-Giles, K.M. (2013a). Genome engineering of Drosophila with the CRISPR RNA-guided Cas9 nuclease. Genetics 194, 1029-1035.
Bruckner, J*., Gratz, S*., Slind, J., Geske, R., Cummings, A., Galindo, S., Donohue, L. and O’Connor-Giles, K. (2012) Fife, a Drosophila Piccolo-RIM homolog, promotes active zone organization and neurotransmitter release. J Neuorsci. 32(48), 17048-58.
*These authors contributed equally.
Babaoglan, A.B., O’Connor-Giles, K.M., Mistry, H. Schickedanz, A., Wilson, B.A. and Skeath, J.B. (2009). Sanpodo: a context-dependent activator and inhibitor of Notch signaling during asymmetric divisions. Development 136, 4089-4098.

O’Connor-Giles, K.M. and Ganetzky, B. (2008). Satellite Signaling at Synapses. Fly 2(5), 259-261.
O’Connor-Giles, K.M., Ho, L.L. and Ganetzky, B. (2008). Nervous wreck interacts with Thickveins and the endocytic machinery to attenuate retrograde BMP growth signaling during synaptic growth. Neuron 58, 507-518.
Eberl, D., Kitamoto, T., Berke, B., O’Connor-Giles, K., Ueda, A., Lee, J., Ruan, H., Engel, J. and Ganetzky, B. (2008). Meeting report: genes, neurons, circuits, and behaviors: highlights of Cold Spring Harbor Meeting on Drosophila neurobiology. Oct 3-7, 2007. Journal of Neurogenetics 22, 3-13.
O’Connor-Giles, K.M. and Skeath, J.B. (2003) Numb inhibits membrane localization of Sanpodo, a four-pass transmembrane protein, to promote asymmetric divisions in Drosophila. Dev Cell 5(2), 231-43.